We also studied patients with other myeloid neoplasms and discovered CALR mutations only in a few patients with RARS-T, a myeloid neoplasm with both myeloproliferative and myelodysplastic features.25 These observations strongly support a causal romantic relationship between CALR mutations and excessive platelet production. Because CALR mutations are found in approximately 73 percent of patients who don’t have mutations of JAK2 and MPL, we believe they fill in the current molecular diagnostic gap in myeloproliferative neoplasms. Altogether, less than 10 percent of our patients with important thrombocythemia or primary myelofibrosis did not have a somatic mutation of JAK2, MPL, or CALR.The CIPAM is normally Mennen’s front end table, which features multiple acquisition models for measuring various physiological parameters. The technical benefit of the CIPAM is founded on many years of development, producing a single board which includes all parameters’ acquisition elements. This permits a development of an array of services which are compact and sizable and yet possess the same powerful abilities of the existing multiple boards which are utilized by other manufacturers.